Oral Presentation 5th International Symposium on Phaeochromocytoma and Paraganglioma 2017

AZEDRA® (iobenguane I 131) in patients with malignant and/or recurrent pheochromocytoma/paraganglioma (PPGL):  final results of a multi-center, open-label, pivotal phase 2b study (#38)

Daniel A. Pryma 1 , Bennett B. Chin 2 , Richard B. Noto 3 , Joseph S. Dillon 4 , Lilja Solnes 5 , Jessica Jensen 6 , Theresa White 6 , Nancy Stambler 6 , Stuart Apfel 6 , Vivien Wong 6 , Camilo Jimenez 7
  1. Perelman School of Medicine at the University of Pennsylvania, Philadelphia, PENNSYLVANIA, United States
  2. Duke University, Durham, NORTH CAROLINA, United States
  3. Warren Alpert Medical School of Brown University, Providence, RHODE ISLAND, United States
  4. University of Iowa Carver College of Medicine, Iowa City, IOWA, United States
  5. Johns Hopkins Medicine, Baltimore, MARYLAND, United States
  6. Progenics Pharmaceuticals, Inc., New York, NEW YORK, United States
  7. University of Texas M. D. Anderson Cancer Center, Houston, TEXAS, United States

Background:  AZEDRA, a high specific activity, proprietary Ultratrace® form of I-131 MIBG, has been developed for the treatment of MIBG-avid metastatic and/or recurrent and/or unresectable PPGL.

Methods:  MIBG-avid patients with PPGL ineligible for curative surgery, failed prior therapy or not candidates for chemotherapy, and on a stable antihypertensive regimen for tumor-related hypertension, were enrolled.  71% of subjects received at least 2 prior lines of therapy.  Subjects received a dosimetric dose (111-222 MBq) followed by up to 2 therapeutic doses, each at 296 MBq/kg to a maximum of 18.5 GBq, approximately 3 months apart.  The primary endpoint measured clinical benefit as defined by the proportion of patients with at least 50% reduction of all antihypertensive medications lasting ≥6 months, and the key secondary endpoint was objective tumor response (RECIST).

Results:  68 patients received at least one therapeutic dose (full analysis; FA).  50 patients received two therapeutic doses (per protocol; PP).  The primary endpoint was met by 25% (95% CI 16%-37%) of FA patients, and 32% (95% CI 21%-46%) of PP patients, achieving pre-specified success criteria of the primary endpoint.  23% and 30% of evaluable FA and PP populations, respectively, achieved best confirmed tumor response of PR.  69% of FA patients and 68% of PP patients achieved best overall response of stable disease.  The most common (≥50%) treatment-emergent adverse events (TEAEs) were nausea, myelosuppression and fatigue.  No acute drug-related hypertensive crises were observed.

Conclusions:  Clinical evidence from this study suggests that treatment with AZEDRA offers meaningful benefits to patients with MIBG-avid malignant, recurrent and/or unresectable PPGL, as measured by reduction in antihypertensive medications, and objective tumor response.  AZEDRA is an effective and well tolerated treatment for an ultra-orphan disease with no approved therapies in the United States.